Since a float is not in the flow, non-positioned block boxes created before and after the float box flow vertically as if the float did not exist. However, the current and subsequent line boxes created next to the float are shortened as necessary to make room for the margin box of the float. A line box is next to a float when there exists a vertical position that satisfies all of these four conditions: (a) at or below the top of the line box, (b) at or above the bottom of the line box, (c) below the top margin edge of the float, and (d) above the bottom margin edge of the float. Note: this means that floats with zero outer height or negative outer height do not shorten line boxes. If a shortened line box is too small to contain any content, then the line box is shifted downward (and its width recomputed) until either some content fits or there are no more floats present. Any content in the current line before a floated box is reflowed in the same line on the other side of the float. In other words, if inline-level boxes are placed on the line before a left float is encountered that fits in the remaining line box space, the left float is placed on that line, aligned with the top of the line box, and then the inline-level boxes already on the line are moved accordingly to the right of the float (the right being the other side of the left float) and vice versa for rtl and right floats.
Brief or mild hypoglycemia produces no lasting effects on the brain, though it can temporarily alter brain responses to additional hypoglycemia. Prolonged, severe hypoglycemia can produce lasting damage of a wide range. This can include impairment of cognitive function, motor control, or even consciousness. The likelihood of permanent brain damage from any given instance of severe hypoglycemia is difficult to estimate, and depends on a multitude of factors such as age, recent blood and brain glucose experience, concurrent problems such as hypoxia , and availability of alternative fuels. It has been frequently found that those Type 1 diabetics found "dead in bed" in the morning after suspected severe hypoglycemia had some underlying coronary pathology that led to an induced fatal heart attack. Recently, several of these individuals found "dead in bed" were wearing Continuous Glucose Monitors, which provided a history of glucose levels prior to the fatal event. It has been found in several cases, that the fatal event was preceded by at least two hours of blood glucose levels under 40 mg/dl, possibly lower as the continuous glucose monitors are not accurate at levels below 40 mg/dl. The individuals failed to respond to the audible alarms produced by the continuous glucose monitor which may have been "alarming" for many hours prior to the fatal event. The vast majority of symptomatic hypoglycemic episodes result in no detectable permanent harm.